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Table 3 Myocardial glucose and lipid metabolism

From: Dietary canolol protects the heart against the deleterious effects induced by the association of rapeseed oil, vitamin E and coenzyme Q10 in the context of a high-fat diet

  C PS R RTC RTCC
pAkt/totAkt 1.64 ± 0.32 1.52 ± 0.25 1.20 ± 0.29 1.29 ± 0.25 1.52 ± 0.23
PDK4 0.13 ± 0.02a 0.28 ± 0.07ab 0.41 ± 0.08b 0.63 ± 0.09c 0.34 ± 0.03b
TG 20 ± 1a 25 ± 2ab 25 ± 2ab 31 ± 4b 24 ± 2a
DG 1.4 ± 0.2ac 0.9 ± 0.09b 1.3 ± 0.1cd 1.1 ± 0.1bd 0.9 ± 0.1b
NEFA 1.2 ± 0.2a 1.1 ± 0.1ab 1.1 ± 0.1ab 0.9 ± 0.1b 0.9 ± 0.1b
Chol 3.2 ± 0.3a 2.7 ± 0.3ab 2.5 ± 0.1b 2.4 ± 0.1b 2.4 ± 0.1b
CE 2.6 ± 0.6 2.3 ± 0.6 1.1 ± 0.2 1.8 ± 0.4 2.0 ± 0.6
PL 70 ± 3 68 ± 2 65 ± 4 63 ± 4 70 ± 3
  1. C control rats, PS rats fed a palm oil/sunflower oil mixture, R rats fed rapeseed oil, RTC rats fed rapeseed oil enriched with α-tocopherol and coenzyme Q10, RTCC rats fed RTC plus canolol, pAkt/totAKt phosphorylated protein kinase B-to-total protein kinase B ratio, PDK4 pyruvate dehydrogenase kinase-4 mRNA expression, TG, DG, NEFA, Chol, CE, PL amounts of triglycerides, diacylglycerols, non-esterified fatty acids, cholesterol, cholesterol esters and phospholipids, respectively, in the myocardium. Averages of 12 rats per group. Lipid amounts are expressed in mg/g of heart weight
  2. a, b, c, dmeans in a row without a common letter are significantly different