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Table 2 The association of dietary nitrate, nitrite and NOx intakes with the incidence of CVD events after 6.7 years of follow-up: Tehran Lipid and Glucose Study 2006–2008 to 2014 (n = 2369)

From: Total antioxidant capacity of the diet modulates the association between habitual nitrate intake and cardiovascular events: A longitudinal follow-up in Tehran Lipid and Glucose Study

  NO3 (≥ 430 mg/d) NO2 (≥ 8.9 mg/d) NOx (≥ 439 mg/d)
< Median dietary TAC 21/332 a 10/309 21/443
Crude 2.89 (1.55–5.37) 0.95 (0.46–1.95) 1.21 (0.64–2.25)
Adjusted b 3.28 (1.54–6.99) 0.64 (0.25–1.62) 0.91 (0.41–1.96)
≥ Median dietary TAC 30/853 32/876 31/742
Crude 1.25 (0.59–2.64) 1.56 (0.69–3.54) 1.42 (0.75–2.70)
Adjusted1 1.10 (0.46–2.61) 2.14 (0.84–5.45) 1.19 (0.54–2.62)
  1. Hazards ratio (95% CI); Cox proportional hazards regression models were used
  2. NO3, NO2, and NOx were included as dichotomous variables (< and ≥ median intakes). Median intake of dietary nitrate, nitrite and nitrate+nitrite was 430, 8.9, and 439 mg/d, respectively
  3. Median dietary TAC was 1284 μmol TE/100 g
  4. TAC total antioxidant capacity; NO3, Nitrate; NO2, Nitrite; NOx, NO3 + NO2
  5. a n (event)/N (total)
  6. b Adjusted for dietary total energy intakes (kcal/d), total fat (g/d), dietary fiber (g/d) and cardiovascular disease risk score