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Table 2 The association of dietary nitrate, nitrite and NOx intakes with the incidence of CVD events after 6.7 years of follow-up: Tehran Lipid and Glucose Study 2006–2008 to 2014 (n = 2369)

From: Total antioxidant capacity of the diet modulates the association between habitual nitrate intake and cardiovascular events: A longitudinal follow-up in Tehran Lipid and Glucose Study

 

NO3−

(≥ 430 mg/d)

NO2−

(≥ 8.9 mg/d)

NOx

(≥ 439 mg/d)

< Median dietary TAC

21/332 a

10/309

21/443

 Crude

2.89 (1.55–5.37)

0.95 (0.46–1.95)

1.21 (0.64–2.25)

 Adjusted b

3.28 (1.54–6.99)

0.64 (0.25–1.62)

0.91 (0.41–1.96)

≥ Median dietary TAC

30/853

32/876

31/742

 Crude

1.25 (0.59–2.64)

1.56 (0.69–3.54)

1.42 (0.75–2.70)

 Adjusted1

1.10 (0.46–2.61)

2.14 (0.84–5.45)

1.19 (0.54–2.62)

  1. Hazards ratio (95% CI); Cox proportional hazards regression models were used
  2. NO3−, NO2−, and NOx were included as dichotomous variables (< and ≥ median intakes). Median intake of dietary nitrate, nitrite and nitrate+nitrite was 430, 8.9, and 439 mg/d, respectively
  3. Median dietary TAC was 1284 μmol TE/100 g
  4. TAC total antioxidant capacity; NO3−, Nitrate; NO2−, Nitrite; NOx, NO3− + NO2−
  5. a n (event)/N (total)
  6. b Adjusted for dietary total energy intakes (kcal/d), total fat (g/d), dietary fiber (g/d) and cardiovascular disease risk score
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Nutrition & Metabolism

ISSN: 1743-7075

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