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Fig. 5 | Nutrition & Metabolism

Fig. 5

From: Lipid deposition pattern and adaptive strategy in response to dietary fat in Chinese perch (Siniperca chuatsi)

Fig. 5

Proposed model of hepatic TG deposition in Chinese perch responding to high-fat intake. L17 represents diets containing of 17% lipid content, which was not benefit for hepatic lipid utilization and metabolism in the present study. In the liver, excess dietary fat-intake could accelerate the esterification of FFAs with the absence of FAs β-oxidation. Indeed, the extra uptake of FFAs could provide the mass Acyl-CoA substrate for lipogenesis via the up-regulation of srebp1. The sharply increased lipogenesis could effectively inhibit gluconeogenesis and aggravate TG deposition in liver although a part of newly-synthesized TG had been delivered to peripheral tissues via VLDL in blood

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