Fig. 2From: Exogenous L-carnitine ameliorates burn-induced cellular and mitochondrial injury of hepatocytes by restoring CPT1 activityEffects of carnitine supplementation on mitochondrial injury in the hepatocytes of burned rats. Representative transmission electron microscopy images of hepatocytes from normal (A), burned rats (B), and carnitine-treated burned rats (C) are shown. Serum levels of OCT (D) and β-HB (E) are shown. The oxygen consumption rate (OCR) of the mitochondria in the liver tissues is shown in (F). Changes in the mitochondrial membrane potential (∆Ψm) in the liver tissues of the normal, burn, and burn + carnitine rats were analyzed via flow cytometry, and the results are shown in (G) and (H). The relative mitochondrial membrane potential in the control is set at 100%. Data are presented as means ± SD. *, P < 0.05; **, P < 0.01; and ***, P < 0.001 versus the control group; ##, P < 0.01 versus the burn groupBack to article page